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Alberico L Catapano, Ian Graham, Guy De Backer, Olov Wiklund, M John Chapman, Heinz Drexel, Arno W Hoes, Catriona S Jennings, Ulf Landmesser, Terje R Pedersen, Željko Reiner, Gabriele Riccardi, Marja-Riita Taskinen, Lale Tokgozoglu, W M Monique Verschuren, Charalambos Vlachopoulos, David A Wood, Jose Luis Zamorano
No abstract text is available yet for this article.
August 27, 2016: European Heart Journal
Liesl Zühlke, Ganesan Karthikeyan, Mark E Engel, Sumathy Rangarajan, Pam Mackie, Blanche Cupido, Katya Mauff, Shofiqul Islam, Rezeen Daniels, Veronica Francis, Stephen Ogendo, Bernard Gitura, Charles Mondo, Emmy Okello, Peter Lwabi, Mohammed M Al-Kebsi, Christopher Hugo-Hamman, Sahar S Sheta, Abraham Haileamlak, Wandimu Daniel, Dejuma Yadeta Goshu, Senbeta G Abdissa, Araya G Desta, Bekele A Shasho, Dufera M Begna, Ahmed ElSayed, Ahmed S Ibrahim, John Musuku, Fidelia Bode-Thomas, Christopher C Yilgwan, Ganiyu A Amusa, Olukemi Ige, Basil Okeahialam, Christopher Sutton, Rajeev Misra, Azza Abul Fadl, Neil Kennedy, Albertino Damasceno, Mahmoud U Sani, Okechukwu S Ogah, Taiwo Olunuga, Huda ElHassan, Ana Olga Mocumbi, Abiodun M Adeoye, Pindile S Mntla, Dike B Ojji, Joseph Mucumbitsi, Koon Teo, Salim Yusuf, Bongani M Mayosi
BACKGROUND: -There are few contemporary data on the mortality and morbidity associated with rheumatic heart disease (RHD) or information on their predictors. We report the two year follow-up of individuals with RHD from 14 low and middle income countries in Africa and Asia. METHODS: -Between January 2010 and November 2012, we enrolled 3343 patients from 25 centers in 14 countries and followed them for two years to assess mortality, congestive heart failure (CHF), stroke or transient ischemic attack (TIA), recurrent acute rheumatic fever (ARF), and infective endocarditis (IE)...
October 4, 2016: Circulation
Jarle Jortveit, Elisabeth Leirgul, Leif Eskedal, Gottfried Greve, Tatiana Fomina, Gaute Døhlen, Grethe S Tell, Sigurd Birkeland, Nina Øyen, Henrik Holmstrøm
BACKGROUND: Ventricular septal defects (VSDs) are the most common congenital heart defects (CHDs). Previous studies indicate an increased risk of endocarditis, aortic regurgitation, left ventricular outflow tract obstructions, pulmonary hypertension, arrhythmias and sudden death in patients with isolated VSDs. The present nationwide cohort study reports mortality and cardiac complications requiring hospitalisation or intervention in children with isolated VSDs. METHODS AND RESULTS: Medical information concerning all 943 871 live births in Norway in 1994-2009 was retrieved from the Medical Birth Registry of Norway, the Cardiovascular Disease in Norway project, the Oslo University Hospital's Clinical Registry of Congenital Heart Defects and the Norwegian Cause of Death Registry...
September 2016: Archives of Disease in Childhood
Therese M Giglia, M Patricia Massicotte, James S Tweddell, Robyn J Barst, Mary Bauman, Christopher C Erickson, Timothy F Feltes, Elyse Foster, Kathleen Hinoki, Rebecca N Ichord, Jacqueline Kreutzer, Brian W McCrindle, Jane W Newburger, Sarah Tabbutt, Jane L Todd, Catherine L Webb
No abstract text is available yet for this article.
December 17, 2013: Circulation
J Shores, K R Berger, E A Murphy, R E Pyeritz
BACKGROUND: The aortic root enlarges progressively in Marfan's syndrome, and this enlargement is associated with aortic regurgitation and dissection. Long-term treatment with beta-adrenergic blockade, by reducing the impulse (i.e., the rate of pressure change in the aortic root) of left ventricular ejection and the heart rate, may protect the aortic root. METHODS: We conducted an open-label, randomized trial of propranolol in adolescent and adult patients with classic Marfan's syndrome (32 treated and 38 untreated [control] patients)...
May 12, 1994: New England Journal of Medicine
Adi Leiba, Gilad Twig, Hagai Levine, Nehama Goldberger, Arnon Afek, Ari Shamiss, Estela Derazne, Dorit Tzur, Ziona Haklai, Jeremy D Kark
BACKGROUND: The effect of early hypertension on midlife cardiovascular (CV) mortality remains controversial. We assessed the association of established hypertension in late adolescence with subsequent CV mortality. METHODS: Of 2,298,130 Israeli adolescents (60% males; age 17.4 ± 0.3 years) who underwent a compulsory medical examination prior to military service between 1967 and 2010, 8720 teenagers (0.4%) were formally diagnosed with persistent hypertension...
March 2016: Pediatric Nephrology: Journal of the International Pediatric Nephrology Association
Geng Zong, Alisa Gao, Frank B Hu, Qi Sun
BACKGROUND: Current findings on associations between whole grain (WG) intake and mortality are inconsistent and have not been summarized by meta-analysis. METHODS AND RESULTS: We searched for prospective cohort studies reporting associations between WG intake and mortality from all causes, cardiovascular disease (CVD), and cancer through February 2016 in Medline, Embase, and, and we further included unpublished results from National Health and Nutrition Examination Survey (NHANES) III and NHANES 1999 to 2004...
June 14, 2016: Circulation
Gene F Kwan, Bongani M Mayosi, Ana O Mocumbi, J Jaime Miranda, Majid Ezzati, Yogesh Jain, Gisela Robles, Emelia J Benjamin, S V Subramanian, Gene Bukhman
The poorest billion people are distributed throughout the world, though most are concentrated in rural sub-Saharan Africa and South Asia. Cardiovascular disease (CVD) data can be sparse in low- and middle-income countries beyond urban centers. Despite this urban bias, CVD registries from the poorest countries have long revealed a predominance of nonatherosclerotic stroke, hypertensive heart disease, nonischemic and Chagas cardiomyopathies, rheumatic heart disease, and congenital heart anomalies, among others...
June 14, 2016: Circulation
Soujanya Bogarapu, Nelangi M Pinto, Susan P Etheridge, Xiaoming Sheng, Kirk N Liesemer, Paul C Young, Elizabeth V Saarel
Current guidelines recommend that all neonates with Down syndrome (DS) be screened for congenital heart disease (CHD) with an echocardiogram. We sought to determine the effectiveness of a more accessible and less expensive screening strategy consisting of physical examination, electrocardiogram (ECG), and chest X-ray. The Intermountain Healthcare Enterprise Data Warehouse was used to identify infants with a positive karyotype for DS who were born between January 1, 2000, and June 30, 2012. Infants with the results of an echocardiogram, physical examination, ECG, and chest X-ray documented at age ≤6 months were included...
October 2016: Pediatric Cardiology
Brady S Moffett, Joshua M Garrison, Aimee Hang, Shaine A Morris, Rocky Tsang, Kimberly Dinh, Pamela Griffiths, Ronald Bronicki, Paul A Checchia
Data regarding availability of prostaglandin E1 (PGE) and its impact on the stabilization, transport, critical care course, and surgical outcome of infants with ductal-dependent congenital heart disease in the current pediatric healthcare environment are unknown. We sought to determine the proportion of hospitals in Texas that stock PGE and to investigate associations between PGE availability and clinical outcomes. All birth institutions listed with the Texas Department of Health and Human Services were contacted to determine PGE availability as of 2011...
February 2016: Pediatric Cardiology
Robert A Levine, Albert A Hagége, Daniel P Judge, Muralidhar Padala, Jacob P Dal-Bianco, Elena Aikawa, Jonathan Beaudoin, Joyce Bischoff, Nabila Bouatia-Naji, Patrick Bruneval, Jonathan T Butcher, Alain Carpentier, Miguel Chaput, Adrian H Chester, Catherine Clusel, Francesca N Delling, Harry C Dietz, Christian Dina, Ronen Durst, Leticia Fernandez-Friera, Mark D Handschumacher, Morten O Jensen, Xavier P Jeunemaitre, Hervé Le Marec, Thierry Le Tourneau, Roger R Markwald, Jean Mérot, Emmanuel Messas, David P Milan, Tui Neri, Russell A Norris, David Peal, Maelle Perrocheau, Vincent Probst, Michael Pucéat, Nadia Rosenthal, Jorge Solis, Jean-Jacques Schott, Ehud Schwammenthal, Susan A Slaugenhaupt, Jae-Kwan Song, Magdi H Yacoub
Mitral valve disease is a frequent cause of heart failure and death. Emerging evidence indicates that the mitral valve is not a passive structure, but--even in adult life--remains dynamic and accessible for treatment. This concept motivates efforts to reduce the clinical progression of mitral valve disease through early detection and modification of underlying mechanisms. Discoveries of genetic mutations causing mitral valve elongation and prolapse have revealed that growth factor signalling and cell migration pathways are regulated by structural molecules in ways that can be modified to limit progression from developmental defects to valve degeneration with clinical complications...
December 2015: Nature Reviews. Cardiology
Leo Lopez, Steven D Colan, Peter C Frommelt, Gregory J Ensing, Kathleen Kendall, Adel K Younoszai, Wyman W Lai, Tal Geva
No abstract text is available yet for this article.
May 2010: Journal of the American Society of Echocardiography
Wyman W Lai, Tal Geva, Girish S Shirali, Peter C Frommelt, Richard A Humes, Michael M Brook, Ricardo H Pignatelli, Jack Rychik
No abstract text is available yet for this article.
December 2006: Journal of the American Society of Echocardiography
Hmwe H Kyu, Christine Pinho, Joseph A Wagner, Jonathan C Brown, Amelia Bertozzi-Villa, Fiona J Charlson, Luc Edgar Coffeng, Lalit Dandona, Holly E Erskine, Alize J Ferrari, Christina Fitzmaurice, Thomas D Fleming, Mohammad H Forouzanfar, Nicholas Graetz, Caterina Guinovart, Juanita Haagsma, Hideki Higashi, Nicholas J Kassebaum, Heidi J Larson, Stephen S Lim, Ali H Mokdad, Maziar Moradi-Lakeh, Shaun V Odell, Gregory A Roth, Peter T Serina, Jeffrey D Stanaway, Awoke Misganaw, Harvey A Whiteford, Timothy M Wolock, Sarah Wulf Hanson, Foad Abd-Allah, Semaw Ferede Abera, Laith J Abu-Raddad, Fadia S AlBuhairan, Azmeraw T Amare, Carl Abelardo T Antonio, Al Artaman, Suzanne L Barker-Collo, Lope H Barrero, Corina Benjet, Isabela M Bensenor, Zulfiqar A Bhutta, Boris Bikbov, Alexandra Brazinova, Ismael Campos-Nonato, Carlos A Castañeda-Orjuela, Ferrán Catalá-López, Rajiv Chowdhury, Cyrus Cooper, John A Crump, Rakhi Dandona, Louisa Degenhardt, Robert P Dellavalle, Samath D Dharmaratne, Emerito Jose A Faraon, Valery L Feigin, Thomas Fürst, Johanna M Geleijnse, Bradford D Gessner, Katherine B Gibney, Atsushi Goto, David Gunnell, Graeme J Hankey, Roderick J Hay, John C Hornberger, H Dean Hosgood, Guoqing Hu, Kathryn H Jacobsen, Sudha P Jayaraman, Panniyammakal Jeemon, Jost B Jonas, André Karch, Daniel Kim, Sungroul Kim, Yoshihiro Kokubo, Barthelemy Kuate Defo, Burcu Kucuk Bicer, G Anil Kumar, Anders Larsson, Janet L Leasher, Ricky Leung, Yongmei Li, Steven E Lipshultz, Alan D Lopez, Paulo A Lotufo, Raimundas Lunevicius, Ronan A Lyons, Marek Majdan, Reza Malekzadeh, Taufiq Mashal, Amanda J Mason-Jones, Yohannes Adama Melaku, Ziad A Memish, Walter Mendoza, Ted R Miller, Charles N Mock, Joseph Murray, Sandra Nolte, In-Hwan Oh, Bolajoko Olubukunola Olusanya, Katrina F Ortblad, Eun-Kee Park, Angel J Paternina Caicedo, Scott B Patten, George C Patton, David M Pereira, Norberto Perico, Frédéric B Piel, Suzanne Polinder, Svetlana Popova, Farshad Pourmalek, D Alex Quistberg, Giuseppe Remuzzi, Alina Rodriguez, David Rojas-Rueda, Dietrich Rothenbacher, David H Rothstein, Juan Sanabria, Itamar S Santos, David C Schwebel, Sadaf G Sepanlou, Amira Shaheen, Rahman Shiri, Ivy Shiue, Vegard Skirbekk, Karen Sliwa, Chandrashekhar T Sreeramareddy, Dan J Stein, Timothy J Steiner, Lars Jacob Stovner, Bryan L Sykes, Karen M Tabb, Abdullah Sulieman Terkawi, Alan J Thomson, Andrew L Thorne-Lyman, Jeffrey Allen Towbin, Kingsley Nnanna Ukwaja, Tommi Vasankari, Narayanaswamy Venketasubramanian, Vasiliy Victorovich Vlassov, Stein Emil Vollset, Elisabete Weiderpass, Robert G Weintraub, Andrea Werdecker, James D Wilkinson, Solomon Meseret Woldeyohannes, Charles D A Wolfe, Yuichiro Yano, Paul Yip, Naohiro Yonemoto, Seok-Jun Yoon, Mustafa Z Younis, Chuanhua Yu, Maysaa El Sayed Zaki, Mohsen Naghavi, Christopher J L Murray, Theo Vos
IMPORTANCE: The literature focuses on mortality among children younger than 5 years. Comparable information on nonfatal health outcomes among these children and the fatal and nonfatal burden of diseases and injuries among older children and adolescents is scarce. OBJECTIVE: To determine levels and trends in the fatal and nonfatal burden of diseases and injuries among younger children (aged <5 years), older children (aged 5-9 years), and adolescents (aged 10-19 years) between 1990 and 2013 in 188 countries from the Global Burden of Disease (GBD) 2013 study...
March 2016: JAMA Pediatrics
Yehuda Adler, Philippe Charron, Massimo Imazio, Luigi Badano, Gonzalo Barón-Esquivias, Jan Bogaert, Antonio Brucato, Pascal Gueret, Karin Klingel, Christos Lionis, Bernhard Maisch, Bongani Mayosi, Alain Pavie, Arsen D Ristić, Manel Sabaté Tenas, Petar Seferovic, Karl Swedberg, Witold Tomkowski, Stephan Achenbach, Stefan Agewall, Nawwar Al-Attar, Juan Angel Ferrer, Michael Arad, Riccardo Asteggiano, Héctor Bueno, Alida L P Caforio, Scipione Carerj, Claudio Ceconi, Arturo Evangelista, Frank Flachskampf, George Giannakoulas, Stephan Gielen, Gilbert Habib, Philippe Kolh, Ekaterini Lambrinou, Patrizio Lancellotti, George Lazaros, Ales Linhart, Philippe Meurin, Koen Nieman, Massimo F Piepoli, Susanna Price, Jolien Roos-Hesselink, François Roubille, Frank Ruschitzka, Jaume Sagristà Sauleda, Miguel Sousa-Uva, Jens Uwe Voigt, Jose Luis Zamorano
No abstract text is available yet for this article.
November 7, 2015: European Heart Journal
Adriana H Tremoulet, Sonia Jain, Preeti Jaggi, Susan Jimenez-Fernandez, Joan M Pancheri, Xiaoying Sun, John T Kanegaye, John P Kovalchin, Beth F Printz, Octavio Ramilo, Jane C Burns
BACKGROUND: Kawasaki disease, the most common cause of acquired heart disease in developed countries, is a self-limited vasculitis that is treated with high doses of intravenous immunoglobulin. Resistance to intravenous immunoglobulin in Kawasaki disease increases the risk of coronary artery aneurysms. We assessed whether the addition of infliximab to standard therapy (intravenous immunoglobulin and aspirin) in acute Kawasaki disease reduces the rate of treatment resistance. METHODS: We undertook a phase 3, randomised, double-blind, placebo-controlled trial in two children's hospitals in the USA to assess the addition of infliximab (5 mg per kg) to standard therapy...
May 17, 2014: Lancet
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