We have located links that may give you full text access.
JOURNAL ARTICLE
RETRACTION OF PUBLICATION
Retraction.
Cancer 2016 October 16
Retraction: "Notch-1 down-regulation by curcumin is associated with the inhibition of cell growth and the induction of apoptosis in pancreatic cancer cells" by Wang Z, Zhang Y, Banerjee S, Li Y, Sarkar FH. The above article, published in Wiley Online Library (wileyonlinelibrary.com), has been retracted by agreement amongst the journal Editor in Chief, Fadlo R. Khuri; Wiley Periodicals, Inc.; and the American Cancer Society following an investigation by Wayne State University into the research activities of the first and last authors. The investigation found clear evidence that the 4-lane Rb bands for Figures 4A, 4B, 5D, and 6D are duplicated and manipulated and re-labeled. The investigation further found no evidence that the 4-lane Rb bands correspond to the assays above them. In addition, the investigation found clear evidence that the 2-lane Rb bands for Figure 4C are duplicated and manipulated and re-labeled Rb bands. The investigation further found no evidence that the 2-lane Rb bands correspond to the "supershift" assays they accompany. There is no evidence that these loading control assays were run at all. The investigation also found that the β-actin bands image in Figure 3D and 5A are re-labeled and manipulated copies of the β-actin bands image in Figure 3C of Int J Cancer. 2006 Apr 15;118(8):1930-6. Finally, the investigation found that the β-actin bands image in Figure 6A is a re-labeled and manipulated copy of Figure 3A of Int J Cancer. 2006 Apr 15;118(8):1930-6, representing two very different experiments. The investigation committee concluded that this undermined the scientific basis of the publication, that no credible replacement data were available, and advised that the publication should be retracted.
REFERENCES: Wang, Z., Zhang, Y., Banerjee, S., Li, Y. and Sarkar, F. H. (2006), Notch-1 down-regulation by curcumin is associated with the inhibition of cell growth and the induction of apoptosis in pancreatic cancer cells. Cancer, 106: 2503-2513. doi: 10.1002/cncr.21904 Wang, Z., Zhang, Y., Banerjee, S., Li, Y. and Sarkar, F. H. (2006), Inhibition of nuclear factor κb activity by genistein is mediated via Notch-1 signaling pathway in pancreatic cancer cells. Int. J. Cancer, 118: 1930-1936. doi: 10.1002/ijc.21589.
REFERENCES: Wang, Z., Zhang, Y., Banerjee, S., Li, Y. and Sarkar, F. H. (2006), Notch-1 down-regulation by curcumin is associated with the inhibition of cell growth and the induction of apoptosis in pancreatic cancer cells. Cancer, 106: 2503-2513. doi: 10.1002/cncr.21904 Wang, Z., Zhang, Y., Banerjee, S., Li, Y. and Sarkar, F. H. (2006), Inhibition of nuclear factor κb activity by genistein is mediated via Notch-1 signaling pathway in pancreatic cancer cells. Int. J. Cancer, 118: 1930-1936. doi: 10.1002/ijc.21589.
Full text links
Related Resources
Trending Papers
Heart failure with preserved ejection fraction: diagnosis, risk assessment, and treatment.Clinical Research in Cardiology : Official Journal of the German Cardiac Society 2024 April 12
Proximal versus distal diuretics in congestive heart failure.Nephrology, Dialysis, Transplantation 2024 Februrary 30
World Health Organization and International Consensus Classification of eosinophilic disorders: 2024 update on diagnosis, risk stratification, and management.American Journal of Hematology 2024 March 30
Efficacy and safety of pharmacotherapy in chronic insomnia: A review of clinical guidelines and case reports.Mental Health Clinician 2023 October
Get seemless 1-tap access through your institution/university
For the best experience, use the Read mobile app
All material on this website is protected by copyright, Copyright © 1994-2024 by WebMD LLC.
This website also contains material copyrighted by 3rd parties.
By using this service, you agree to our terms of use and privacy policy.
Your Privacy Choices
You can now claim free CME credits for this literature searchClaim now
Get seemless 1-tap access through your institution/university
For the best experience, use the Read mobile app