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Mitochondrial biogenesis meets chemoresistance in BRAF-mutant melanoma.

Lawrence W Wu, Gao Zhang, Meenhard Herlyn
Molecular & Cellular Oncology 2016 July
The acquisition of resistance to current mitogen activated protein kinase (MAPK) inhibitors in B-Raf proto-oncogene, serine/threonine kinase (BRAF) mutant melanoma is almost inevitable. Our recent findings identify therapy-induced mitochondrial biogenesis (MitoBiogenesis) and aberrant tumor bioenergetics as therapeutic escape mechanisms and offer a rational combinatorial strategy to further improve the efficacy of MAPK inhibitors.

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