Add like
Add dislike
Add to saved papers

Mechanisms of the neuromuscular blocking activity of the aminoglycoside antibiotic streptomycin.

While increasing the spontaneous quantal transmitter release in frog neuromuscular junctions, streptomycin blocked the evoked transmitter release by decreasing the quantal content (m). The quantal parameter of release n (number of release sites) was significantly decreased, while p (probability of release through transmitter activation) increased. Streptomycin competes with calcium ions for the membrane release sites while the intracellular mechanisms of release remain unaffected. The data show a good fit to the two-step hypothesis of quantal transmitter release recently developed on the basis of ouabain actions.

Full text links

We have located links that may give you full text access.
Can't access the paper?
Try logging in through your university/institutional subscription. For a smoother one-click institutional access experience, please use our mobile app.

For the best experience, use the Read mobile app

Mobile app image

Get seemless 1-tap access through your institution/university

For the best experience, use the Read mobile app

All material on this website is protected by copyright, Copyright © 1994-2024 by WebMD LLC.
This website also contains material copyrighted by 3rd parties.

By using this service, you agree to our terms of use and privacy policy.

Your Privacy Choices Toggle icon

You can now claim free CME credits for this literature searchClaim now

Get seemless 1-tap access through your institution/university

For the best experience, use the Read mobile app