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icuaw and electric muscle stimulation

M Goll, T Wollersheim, K Haas, R Moergeli, J Malleike, F Nehls, K Reiher, N Carbon, G Sonomoya, S Weber-Carstens
No abstract text is available yet for this article.
December 2015: Intensive Care Medicine Experimental
Paul Nardelli, Jacob A Vincent, Randall Powers, Tim C Cope, Mark M Rich
The mechanisms by which sepsis triggers intensive care unit acquired weakness (ICUAW) remain unclear. We previously identified difficulty with motor unit recruitment in patients as a novel contributor to ICUAW. To study the mechanism underlying poor recruitment of motor units we used the rat cecal ligation and puncture model of sepsis. We identified striking dysfunction of alpha motor neurons during repetitive firing. Firing was more erratic, and often intermittent. Our data raised the possibility that reduced excitability of motor neurons was a significant contributor to weakness induced by sepsis...
August 2016: Experimental Neurology
Epameinondas Angelopoulos, Eleftherios Karatzanos, Stavros Dimopoulos, Georgios Mitsiou, Christos Stefanou, Irini Patsaki, Anastasia Kotanidou, Christina Routsi, George Petrikkos, Serafeim Nanas
BACKGROUND: Intensive care unit-acquired weakness (ICUAW) is a common complication, associated with significant morbidity. Neuromuscular electrical stimulation (NMES) has shown promise for prevention. NMES acutely affects skeletal muscle microcirculation; such effects could mediate the favorable outcomes. However, optimal current characteristics have not been defined. This study aimed to compare the effects on muscle microcirculation of a single NMES session using medium and high frequency currents...
2013: Annals of Intensive Care
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