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Feeding and eating disorders

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https://www.readbyqxmd.com/read/27904991/eat-up%C3%A2-family-centered-feeding-intervention-to-promote-food-acceptance-and-decrease-challenging-behaviors-a-single-case-experimental-design-replicated-across-three-families-of-children-with-autism-spectrum-disorder
#1
Joanna Cosbey, Deirdre Muldoon
This study evaluated the effectiveness of a family-centered feeding intervention, Easing Anxiety Together with Understanding and Perseverance (EAT-UP™), for promoting food acceptance of children with autism spectrum disorder at home. A concurrent multiple-baseline design was used with systematic replication across three families. Baseline was followed by an 'Intervention-Coaching' phase and then an 'Intervention-Independent' phase. Using direct observation and pre- and post-intervention questionnaires, data on acceptance of less preferred foods and challenging mealtime behaviors were collected...
November 30, 2016: Journal of Autism and Developmental Disorders
https://www.readbyqxmd.com/read/27889494/maternal-history-of-eating-disorders-diet-quality-during-pregnancy-and-infant-feeding
#2
Anh N Nguyen, Lisanne M de Barse, Henning Tiemeier, Vincent W V Jaddoe, Oscar H Franco, Pauline W Jansen, Trudy Voortman
We studied associations of maternal history of eating disorders (EDs) with diet quality of pregnant women and their infants, and breastfeeding practices. We included 6196 mother-child pairs from Generation R, a population-based cohort in the Netherlands. Maternal history of lifetime EDs was assessed during pregnancy with a questionnaire. Dietary intake during pregnancy and in infancy was assessed with food-frequency questionnaires and diet quality scores were calculated, reflecting adherence to dietary guidelines...
November 23, 2016: Appetite
https://www.readbyqxmd.com/read/27884425/the-eating-disorder-associated-hdac4-a778t-mutation-alters-feeding-behaviors-in-female-mice
#3
Michael Lutter, Michael Z Khan, Kenji Satio, Kevin C Davis, Ian J Kidder, Latisha McDaniel, Benjamin W Darbro, Andrew A Pieper, Huxing Cui
BACKGROUND: While eating disorders (EDs) are thought to result from a combination of environmental and psychological stressors superimposed on genetic vulnerability, the neurobiological basis of EDs remains incompletely understood. We recently reported that a rare missense mutation in the gene for the transcriptional repressor histone deacetylase 4 (HDAC4) is associated with the risk of developing an ED in humans. METHODS: To understand the biological consequences of this missense mutation, we created transgenic mice carrying this mutation by introducing the alanine to threonine mutation at position 778 of mouse Hdac4 (corresponding to position 786 of the human protein)...
October 13, 2016: Biological Psychiatry
https://www.readbyqxmd.com/read/27879968/can-urinary-indolylacroylglycine-iag-levels-be-used-to-determine-whether-children-with-autism-will-benefit-from-dietary-intervention
#4
Julie Wilson, Barry Wright, Sandra Jost, Robert Smith, Helen Pearce, Sally Richardson
BACKGROUND: An increase in urinary indolyl-3-acryloylglycine (IAG) has been reported in children with ASD who suffer with bowel problems in comparison to ASD children without gastrointestinal (GI) problems. The case for dietary intervention for ASD children with GI symptoms might be strengthened were such a difference to be autism-specific. METHODS: Quantitative analysis of urinary IAG levels was performed for 53 children on the autism spectrum and 146 age-matched controls...
November 23, 2016: Pediatric Research
https://www.readbyqxmd.com/read/27862127/the-impact-of-dsm-5-on-eating-disorder-diagnoses
#5
Megen Vo, Erin C Accurso, Andrea B Goldschmidt, Daniel Le Grange
OBJECTIVE: Eating disorder diagnostic criteria were revised from the fourth to the fifth edition of the Diagnostic and Statistical Manual of Mental Disorders (DSM-IV and -5, respectively). This study examines the impact of these revisions on rates of eating disorder diagnoses in treatment-seeking youth. METHOD: Participants were 651 youth, ages 7-18 years, presenting to an outpatient eating disorders program who met criteria for a DSM-IV eating disorder diagnosis on intake...
November 12, 2016: International Journal of Eating Disorders
https://www.readbyqxmd.com/read/27861553/activity-based-anorexia-alters-the-expression-of-bdnf-transcripts-in-the-mesocorticolimbic-reward-circuit
#6
Emily V Ho, Stephanie J Klenotich, Matthew S McMurray, Stephanie C Dulawa
Anorexia nervosa (AN) is a complex eating disorder with severe dysregulation of appetitive behavior. The activity-based anorexia (ABA) paradigm is an animal model in which rodents exposed to both running wheels and scheduled feeding develop aspects of AN including paradoxical hypophagia, dramatic weight loss, and hyperactivity, while animals exposed to only one condition maintain normal body weight. Brain-derived neurotrophic factor (BDNF), an activity-dependent modulator of neuronal plasticity, is reduced in the serum of AN patients, and is a known regulator of feeding and weight maintenance...
2016: PloS One
https://www.readbyqxmd.com/read/27859458/children-of-parents-with-bed-have-more-eating-behavior-disturbance-than-children-of-parents-with-obesity-or-healthy-weight
#7
Janet A Lydecker, Carlos M Grilo
OBJECTIVE: A limited literature suggests an association between parental eating disorders and child eating-disorder behaviors although this research has focused primarily on restrictive-type eating disorders and very little is known about families with binge-eating disorder (BED). METHODS: The current study focused on parents (N = 331; 103 fathers and 226 mothers), comparing parents with core features of BED (n = 63) to parents with obesity and no eating disorder (OB; n = 85) and parents with healthy-weight and no eating disorder (HW; n = 183)...
November 12, 2016: International Journal of Eating Disorders
https://www.readbyqxmd.com/read/27810402/impact-of-intermittent-fasting-on-health-and-disease-processes
#8
REVIEW
Mark P Mattson, Valter D Longo, Michelle Harvie
Humans in modern societies typically consume food at least three times daily, while laboratory animals are fed ad libitum. Overconsumption of food with such eating patterns often leads to metabolic morbidities (insulin resistance, excessive accumulation of visceral fat, etc.), particularly when associated with a sedentary lifestyle. Because animals, including humans, evolved in environments where food was relatively scarce, they developed numerous adaptations that enabled them to function at a high level, both physically and cognitively, when in a food-deprived/fasted state...
October 31, 2016: Ageing Research Reviews
https://www.readbyqxmd.com/read/27807162/an-inhibitory-septum-to-lateral-hypothalamus-circuit-that-suppresses-feeding
#9
Patrick Sweeney, Yunlei Yang
: Feeding behavior is orchestrated by neural circuits primarily residing in the hypothalamus and hindbrain. However, the relative influence of cognitive and emotional brain circuits to the feeding circuitry in the hypothalamus and hindbrain remains unclear. Here, using the cell-type selectivity of genetic methods, circuit mapping, and behavior assays, we sought to decipher neural circuits emanating from the septal nucleus to the lateral hypothalamus (LH) that contribute to neural regulation of food intake in mice...
November 2, 2016: Journal of Neuroscience: the Official Journal of the Society for Neuroscience
https://www.readbyqxmd.com/read/27798108/behavioural-abnormalities-in-novel-mouse-model-for-silver-russell-syndrome
#10
Grainne Iseult McNamara, Brittany Ann Davis, Dominic Michael Dwyer, Rosalind M John, Anthony Roger Isles
Silver Russell Syndrome (SRS) syndrome is an imprinting disorder involving low birth weight with complex genetics and diagnostics. Some rare SRS patients carry maternally inherited microduplications spanning the imprinted genes CDKN1C, PHLDA2, SLC22A18 and KCNQ1, suggesting that overexpression of one of more of these genes contributes to the SRS phenotype. While this molecular alteration is very rare, feeding difficulties are a very common feature of this condition. Given that SRS children also have very low body mass index, understanding the underpinning biology of the eating disorder is important, as well as potential co-occurring behavioural alterations...
October 24, 2016: Human Molecular Genetics
https://www.readbyqxmd.com/read/27765644/binge-like-intake-of-hfd-attenuates-alcohol-intake-in-rats
#11
Sunil Sirohi, Arriel Van Cleef, Jon F Davis
Binge eating and binge alcohol intake are behavioral manifestations of pathological feeding and alcohol use disorder (AUD), respectively. Binge-feeding and AUD have high comorbidity with other psychiatric disorders such as depression, which could have important implications for the management of these conditions. Importantly, these behaviors share many common features suggesting a singular etiology. However, the nature by which binge-feeding affects the development or maintenance of AUD is unclear. The present study examined the impact of a binge-feeding from a nutritionally complete high-fat diet (HFD) on initiation and maintenance of alcohol intake, anxiolytic behavior and central genetic changes in brain regions that control alcohol-reinforced behaviors...
October 17, 2016: Physiology & Behavior
https://www.readbyqxmd.com/read/27744763/a-review-of-trazodone-use-in-psychiatric-and-medical-conditions
#12
Hani Raoul Khouzam
Trazodone is an antidepressant that is FDA-approved for the treatment of depression. It has been used by mental health and primary care providers for the treatment of multiple psychiatric and medical conditions .This review describes trazodone mechanism of action, formulation, dosage and adverse effects and then summarizes the beneficial effects of trazodone in the treatment of various psychiatric and medical conditions such as major depression, as well non-approved FDA indications such as insomnia,anxiety disorders, posttraumatic stress disorder, obsessive compulsive disorder, feeding and eating disorders, substance use disorders, behavioral disturbances associated with cognitive dysfunction, sexual dysfunction, certain pain conditions, and rehabilitation after acute ischemic stroke...
October 17, 2016: Postgraduate Medicine
https://www.readbyqxmd.com/read/27742391/-early-restrictive-feeding-disorders-quantitative-assessment-of-parent-infant-feeding-interactions
#13
A Bion, T Cascales, S Dubedout, N Bodeau, J P Olives, J P Raynaud
INTRODUCTION: Interest in the study of early feeding disorders (FD) has steadily increased during recent decades. During this period, research described the importance of the transactional relationships and the complex interplay between caregiver and child over time. On the basis of the previous studies, our study tried to explore the associations between the characteristics of the parents and the temperamental characteristics of the infants with early FD. GOALS: A first aim of the present study was to show if parental perception of child temperament (including ability for arousal self-regulation) and parental characteristics (emotional and eating attitudes) are associated with early FD...
October 11, 2016: L'Encéphale
https://www.readbyqxmd.com/read/27711047/the-trace-amine-associated-receptor-1-agonist-ro5256390-blocks-compulsive-binge-like-eating-in-rats
#14
Antonio Ferragud, Adam D Howell, Catherine F Moore, Tina L Ta, Marius C Hoener, Valentina Sabino, Pietro Cottone
Compulsive, binge eating of highly palatable food constitutes a core feature of some forms of obesity and eating disorders, as well as of the recently proposed disorder of food addiction. Trace amine-associated receptor 1 (TAAR1) is a highly conserved G-protein-coupled receptor bound by endogenous trace amines. TAAR1 agonists have been shown to reduce multiple behavioral effects of drugs of abuse through their actions on the mesocorticolimbic system. In this study, we hypothesized that TAAR1 may have a role in compulsive, binge-like eating; we tested this hypothesis by assessing the effects of a TAAR1 agonist, RO5256390, in multiple excessive feeding-related behaviors induced by limiting access to a highly palatable diet in rats...
November 2, 2016: Neuropsychopharmacology: Official Publication of the American College of Neuropsychopharmacology
https://www.readbyqxmd.com/read/27698417/a-cholinergic-basal-forebrain-feeding-circuit-modulates-appetite-suppression
#15
Alexander M Herman, Joshua Ortiz-Guzman, Mikhail Kochukov, Isabella Herman, Kathleen B Quast, Jay M Patel, Burak Tepe, Jeffrey C Carlson, Kevin Ung, Jennifer Selever, Qingchun Tong, Benjamin R Arenkiel
Atypical food intake is a primary cause of obesity and other eating and metabolic disorders. Insight into the neural control of feeding has previously focused mainly on signalling mechanisms associated with the hypothalamus, the major centre in the brain that regulates body weight homeostasis. However, roles of non-canonical central nervous system signalling mechanisms in regulating feeding behaviour have been largely uncharacterized. Acetylcholine has long been proposed to influence feeding owing in part to the functional similarity between acetylcholine and nicotine, a known appetite suppressant...
October 3, 2016: Nature
https://www.readbyqxmd.com/read/27663813/childhood-obesity-global-issues
#16
Maria Rosaria Juli
The eating disorders are a group of situations which are very complex that include abnormal feeding patterns, too much worry about the physical aspect, no real perception of the body image and a strong link between all these factors and the levels of self-esteem (Fairburn & Harrison 2003, Sigel 2008, American Academy of Pediatrics 2010, Dalle Grave 2011). From the '50s of the previous century we had a continuous increase of the Eating Disorders (Dalle Grave 2011). Indeed, in the National Program of the Prevention is stated that: "The spread of the eating disorders is very fast and relevant; there is no other disease with the same propagation and that looks like a real social epidemic" (The Ministry of Health 2010)...
September 2016: Psychiatria Danubina
https://www.readbyqxmd.com/read/27649625/eating-disorders-from-bench-to-bedside-and-back
#17
Silvana Gaetani, Adele Romano, Gustavo Provensi, Valdo Ricca, Thomas Lutz, Maria Beatrice Passani
The central nervous system and viscera constitute a functional ensemble, the gut-brain axis, that allows bidirectional information flow that contributes to the control of feeding behavior based not only on the homeostatic, but also on the hedonic aspects of food intake. The prevalence of eating disorders, such as anorexia nervosa, binge eating and obesity, poses an enormous clinical burden, and involves an ever-growing percentage of the population worldwide. Clinical and preclinical research is constantly adding new information to the field and orienting further studies with the aim of providing a foundation for developing more specific and effective treatment approaches to pathological conditions...
September 21, 2016: Journal of Neurochemistry
https://www.readbyqxmd.com/read/27594387/characteristics-of-avoidant-restrictive-food-intake-disorder-in-a-cohort-of-adult-patients
#18
Yoshikatsu Nakai, Kazuko Nin, Shun'ichi Noma, Satoshi Teramukai, Stephen A Wonderlich
OBJECTIVE: To assess and compare clinical symptoms and psychometric analysis of adult patients with avoidant/restrictive food intake disorder (ARFID) with those with anorexia nervosa (AN). METHOD: We completed a retrospective review of adult patients with a feeding and eating disorder assessed between 1990 and 2005 that qualified for a diagnosis of ARFID. Patients with ARFID were compared with those with AN, with respect to the demographics, clinical symptoms and psychometric analysis...
September 4, 2016: European Eating Disorders Review: the Journal of the Eating Disorders Association
https://www.readbyqxmd.com/read/27573908/reassessment-of-patients-with-eating-disorders-after-moving-from-dsm-iv-towards-dsm-5-a-retrospective-study-in-a-clinical-sample
#19
Malvina Gualandi, Marzia Simoni, Emilia Manzato, Giovanni Scanelli
PURPOSE: To compare the relative prevalence of eating disorders moving from DSM-IV to DSM-5, and to reassess the overall medical impairment in the revised diagnostic classes. METHODS: We applied DSM-5 to 206 patients (age 15-56 years) previously studied and classified according to DSM-IV. Medical impairment was classified as low, medium, or high, based on a cumulative score of clinical severity (SCS), computed as the sum of specific weights assigned to different pathological conditions and their ascertained prognostic impact...
August 29, 2016: Eating and Weight Disorders: EWD
https://www.readbyqxmd.com/read/27562056/feeding-during-the-resting-phase-causes-profound-changes-in-physiology-and-desynchronization-between-liver-and-muscle-rhythms-of-rats
#20
Anne-Loes Opperhuizen, Dawei Wang, Ewout Foppen, Remi Jansen, Olga Boudzovitch-Surovtseva, Janneke de Vries, Eric Fliers, Andries Kalsbeek
Shiftworkers run an increased risk of developing metabolic disorders, presumably as a result of disturbed circadian physiology. Eating at a time-of-day that is normally dedicated to resting and fasting, may contribute to this association. The hypothalamus is the key brain area that integrates different inputs, including environmental time information from the central biological clock in the suprachiasmatic nuclei, with peripheral information on energy status to maintain energy homeostasis. The orexin system within the lateral hypothalamus is an important output of the suprachiasmatic nuclei involved in the control of sleep/wake behavior and glucose homeostasis, among other functions...
November 2016: European Journal of Neuroscience
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