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alcohol brugada syndrome

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https://www.readbyqxmd.com/read/27186380/intoxication-with-alcohol-an-underestimated-trigger-of-brugada-syndrome
#1
Andrew Achaiah, Neil Andrews
Alcohol intoxication is a potentially under-recognised precipitant of Brugada syndrome. Higher pre-cordial electrocardiogram lead placement increases sensitivity of detecting the Brugada pattern.
May 2016: JRSM Open
https://www.readbyqxmd.com/read/27038845/a-case-of-brugada-syndrome-presenting-with-ventricular-fibrillation-storm-and-prominent-early-repolarization
#2
Chifumi Iizuka, Masahito Sato, Hitoshi Kitazawa, Yoshio Ikeda, Masaaki Okabe, Kiyotaka Kugiyama, Yoshifusa Aizawa
A 21-year-old man developed ventricular fibrillation (VF) while drinking alcohol and was admitted to our hospital. An electrocardiogram (ECG) on admission revealed remarkably prominent slurs on the terminal part of QRS complexes in the left precordial leads and a coved type ST elevation at higher intercostal spaces. After hypothermia therapy, he underwent implantation of an implantable cardioverter-defibrillator (ICD). Standard twelve-lead follow-up ECGs revealed early repolarization pattern and an intermittent coved type ST elevation...
July 2016: Journal of Electrocardiology
https://www.readbyqxmd.com/read/26968993/insight-into-specific-pro-arrhythmic-triggers-in-brugada-and-early-repolarization-syndromes-results-of-long-term-follow-up
#3
Ahmed Karim Talib, Nobuyuki Sato, Takuya Myojo, Eitaro Sugiyama, Naoki Nakagawa, Naka Sakamoto, Yasuko Tanabe, Takayuki Fujino, Toshiharu Takeuchi, Kazumi Akasaka, Hironobu Matsuhashi, Yasuaki Saijo, Yuichiro Kawamura, Atsushi Doi, Naoyuki Hasebe
The pro-arrhythmic triggers in Brugada and early repolarization syndromes (BrS, ERS) have not been analyzed systematically except for case reports. We clinically investigated the circumstances which precede/predispose to arrhythmic events in these syndromes during long-term follow-up. A detailed history from the patients/witnesses was taken to investigate the antecedent events in the last few hours that preceded syncope/ventricular fibrillation (VF); medical records, ECG and blood test from the emergency room (ER) were reviewed...
March 11, 2016: Heart and Vessels
https://www.readbyqxmd.com/read/26940122/screening-high-school-students-in-italy-for-sudden-cardiac-death-prevention-by-using-a-telecardiology-device-a-retrospective-observational-study
#4
Claudio De Lazzari, Igino Genuini, Maria C Gatto, Alessandra Cinque, Massimo Mancone, Alessandra D'Ambrosi, Elisa Silvetti, Antonio Fusto, Domenico M Pisanelli, Francesco Fedele
BACKGROUND: In 2010, an Italian project was launched aimed at using a telecardiology device in order to perform early diagnosis of young students at risk of sudden cardiac death. METHODS: Our retrospective observational study was conducted on a population of 13,016 students, aged between 16 and 19 years, in different Italian regions. It consisted of analysis of data recorded during a telecardiology pilot study. The recorded data were electrocardiograms and data concerning lifestyle habits and family history of cardiovascular diseases...
March 4, 2016: Cardiology in the Young
https://www.readbyqxmd.com/read/25399050/genetic-and-toxicologic-investigation-of-sudden-cardiac-death-in-a-patient-with-arrhythmogenic-right-ventricular-cardiomyopathy-arvc-under-cocaine-and-alcohol-effects
#5
Francesca Cittadini, Nadia De Giovanni, Mireia Alcalde, Sara Partemi, Arnaldo Carbone, Oscar Campuzano, Ramon Brugada, Antonio Oliva
Cocaine and alcohol toxicity is well known, especially when simultaneously abused. These drugs perform both acute and chronic harmfulness, with significant cardiac events such as ventricular arrhythmias, tachycardia, systemic hypertension, acute myocardial infarction, ventricular hypertrophy, and acute coronary syndrome. The present report refers about a patient who died after a documented episode of psychomotor agitation followed by cardiac arrest. At the autopsy investigation, arrhythmogenic right ventricular cardiomyopathy (ARVC) was diagnosed and confirmed by postmortem molecular analysis revealing a mutation in the DSG2 gene...
January 2015: International Journal of Legal Medicine
https://www.readbyqxmd.com/read/24903622/brugada-phenocopy-in-concomitant-ethanol-and-heroin-overdose
#6
Mehdi Rambod, Sherif Elhanafi, Debabrata Mukherjee
Brugada phenocopy describes conditions with Brugada-like ECG pattern but without true congenital Brugada syndrome. We report a case of 44-year-old man with no known medical history who presented with loss of consciousness. Toxicology screening was positive for opiates and high serum alcohol level. His initial ECG showed Brugada type 1 pattern which resolved after several hours of observation and treatment with continuous naloxone infusion. Patient regained his consciousness and disclosed heroin abuse and drinking alcohol...
January 2015: Annals of Noninvasive Electrocardiology
https://www.readbyqxmd.com/read/23365071/alcohol-induced-ventricular-fibrillation-in-a-case-of-brugada-syndrome
#7
Kimie Ohkubo, Toshiko Nakai, Ichiro Watanabe
No abstract text is available yet for this article.
July 2013: Europace: European Pacing, Arrhythmias, and Cardiac Electrophysiology
https://www.readbyqxmd.com/read/18651443/the-management-of-brugada-syndrome-patients
#8
Andrés Ricardo Pérez Riera, Li Zhang, Augusto Hiroshi Uchida, Edgardo Schapachnik, Sergio Dubner, Celso Ferreira
Brugada syndrome is a congenital electrical disorder characterised by the appearance of distinctive QRST-T patterns in the right precordial leads and an increased risk of sudden death (SCD) in young healthy adults. Although chamber enlargement is not apparent in most cases, autopsy and histological investigations have revealed structural abnormalities. The typical Brugada ECG manifestation is often concealed and may be revealed by Class IC anti-arrhythmic agents with the effect of blocking the fast component of sodium channel currents...
2007: Cardiology Journal
https://www.readbyqxmd.com/read/18261498/removing-the-mask
#9
Rory B Weiner, Shepard D Weiner, Peter M Yurchak
No abstract text is available yet for this article.
February 2008: American Journal of Medicine
https://www.readbyqxmd.com/read/17967977/mutation-in-glycerol-3-phosphate-dehydrogenase-1-like-gene-gpd1-l-decreases-cardiac-na-current-and-causes-inherited-arrhythmias
#10
Barry London, Michael Michalec, Haider Mehdi, Xiaodong Zhu, Laurie Kerchner, Shamarendra Sanyal, Prakash C Viswanathan, Arnold E Pfahnl, Lijuan L Shang, Mohan Madhusudanan, Catherine J Baty, Stephen Lagana, Ryan Aleong, Rebecca Gutmann, Michael J Ackerman, Dennis M McNamara, Raul Weiss, Samuel C Dudley
BACKGROUND: Brugada syndrome is a rare, autosomal-dominant, male-predominant form of idiopathic ventricular fibrillation characterized by a right bundle-branch block and ST elevation in the right precordial leads of the surface ECG. Mutations in the cardiac Na+ channel SCN5A on chromosome 3p21 cause approximately 20% of the cases of Brugada syndrome; most mutations decrease inward Na+ current, some by preventing trafficking of the channels to the surface membrane. We previously used positional cloning to identify a new locus on chromosome 3p24 in a large family with Brugada syndrome and excluded SCN5A as a candidate gene...
November 13, 2007: Circulation
https://www.readbyqxmd.com/read/17967976/molecular-and-functional-characterization-of-novel-glycerol-3-phosphate-dehydrogenase-1-like-gene-gpd1-l-mutations-in-sudden-infant-death-syndrome
#11
David W Van Norstrand, Carmen R Valdivia, David J Tester, Kazuo Ueda, Barry London, Jonathan C Makielski, Michael J Ackerman
BACKGROUND: Autopsy-negative sudden unexplained death, including sudden infant death syndrome, can be caused by cardiac channelopathies such as Brugada syndrome (BrS). Type 1 BrS, caused by mutations in the SCN5A-encoded sodium channel, accounts for approximately 20% of BrS. Recently, a novel mutation in the glycerol-3-phosphate dehydrogenase 1-like gene (GPD1-L) disrupted trafficking of SCN5A in a multigenerational family with BrS. We hypothesized that mutations in GPD1-L may be responsible for some cases of sudden unexplained death/sudden infant death syndrome...
November 13, 2007: Circulation
https://www.readbyqxmd.com/read/16610350/therapy-for-the-brugada-syndrome
#12
REVIEW
C Antzelevitch, J M Fish
The Brugada syndrome is a congenital syndrome of sudden cardiac death first described as a new clinical entity in 1992. Electrocardiographically characterized by a distinct coved-type ST segment elevation in the right precordial leads, the syndrome is associated with a high risk for sudden cardiac death in young and otherwise healthy adults, and less frequently in infants and children. The ECG manifestations of the Brugada syndrome are often dynamic or concealed and may be revealed or modulated by sodium channel blockers...
2006: Handbook of Experimental Pharmacology
https://www.readbyqxmd.com/read/15898165/brugada-syndrome-report-of-the-second-consensus-conference
#13
REVIEW
Charles Antzelevitch, Pedro Brugada, Martin Borggrefe, Josep Brugada, Ramon Brugada, Domenico Corrado, Ihor Gussak, Herve LeMarec, Koonlawee Nademanee, Andres Ricardo Perez Riera, Wataru Shimizu, Eric Schulze-Bahr, Hanno Tan, Arthur Wilde
Since its introduction as a clinical entity in 1992, the Brugada syndrome has progressed from being a rare disease to one that is second only to automobile accidents as a cause of death among young adults in some countries. Electrocardiographically characterized by a distinct ST-segment elevation in the right precordial leads, the syndrome is associated with a high risk for sudden cardiac death in young and otherwise healthy adults, and less frequently in infants and children. Patients with a spontaneously appearing Brugada ECG have a high risk for sudden arrhythmic death secondary to ventricular tachycardia/fibrillation...
April 2005: Heart Rhythm: the Official Journal of the Heart Rhythm Society
https://www.readbyqxmd.com/read/15655131/brugada-syndrome-report-of-the-second-consensus-conference-endorsed-by-the-heart-rhythm-society-and-the-european-heart-rhythm-association
#14
REVIEW
Charles Antzelevitch, Pedro Brugada, Martin Borggrefe, Josep Brugada, Ramon Brugada, Domenico Corrado, Ihor Gussak, Herve LeMarec, Koonlawee Nademanee, Andres Ricardo Perez Riera, Wataru Shimizu, Eric Schulze-Bahr, Hanno Tan, Arthur Wilde
Since its introduction as a clinical entity in 1992, the Brugada syndrome has progressed from being a rare disease to one that is second only to automobile accidents as a cause of death among young adults in some countries. Electrocardiographically characterized by a distinct ST-segment elevation in the right precordial leads, the syndrome is associated with a high risk for sudden cardiac death in young and otherwise healthy adults, and less frequently in infants and children. Patients with a spontaneously appearing Brugada ECG have a high risk for sudden arrhythmic death secondary to ventricular tachycardia/fibrillation...
February 8, 2005: Circulation
https://www.readbyqxmd.com/read/10816181/-recurrent-ventricular-fibrillation-during-a-febrile-illness-in-a-patient-with-the-brugada-syndrome
#15
J M González Rebollo, A Hernández Madrid, A García, A García de Castro, A Mejías, C Moro
Different situations have been involved in the origin of ventricular arrhythmic events in patients with the Brugada syndrome such as bradycardia, alcohol consumption and mental stress. We present a 30 year old male with recurrent ventricular fibrillation due to a febrile illness with intense sweating. He had been previously studied at our Unit in 1995 because of an episode of resuscitated cardiac arrest due to ventricular fibrillation. The twelve-lead electrocardiogram showed the typical characteristics of a patient with the Brugada syndrome...
May 2000: Revista Española de Cardiología
https://www.readbyqxmd.com/read/8844303/sustained-monomorphic-ventricular-tachycardia-in-a-patient-with-brugada-syndrome
#16
M Shimada, T Miyazaki, S Miyoshi, K Soejima, S Hori, H Mitamura, S Ogawa
We report a patient with Brugada syndrome who developed sustained monomorphic ventricular tachycardia (SMVT). The patient was a 29-year-old man who experienced recurrent episodes of palpitation and syncope after drinking alcohol. Electrocardiogram showed right bundle branch block and ST-segment elevation in precordial leads V1-3 without Q-Tc prolongation. Organic heart disease and coronary artery disease were excluded by noninvasive and invasive tests. Ventricular fibrillation was induced by the application of a single extra-stimulus to the right ventricular outflow tract...
June 1996: Japanese Circulation Journal
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