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Harm reductio

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https://www.readbyqxmd.com/read/29464993/beyond-infanticide-how-psychological-accounts-of-persons-can-justify-harming-infants
#1
Daniel Rodger, Bruce P Blackshaw, Calum Miller
It is commonly argued that a serious right to life is grounded only in actual, relatively advanced psychological capacities a being has acquired. The moral permissibility of abortion is frequently argued for on these grounds. Increasingly it is being argued that such accounts also entail the permissibility of infanticide, with several proponents of these theories accepting this consequence. We show, however, that these accounts imply the permissibility of even more unpalatable acts than infanticide performed on infants: organ harvesting, live experimentation, sexual interference, and discriminatory killing...
February 21, 2018: New Bioethics: a Multidisciplinary Journal of Biotechnology and the Body
https://www.readbyqxmd.com/read/23637456/killing-fetuses-and-killing-newborns
#2
COMMENT
Ezio Di Nucci
The argument for the moral permissibility of killing newborns is a challenge to liberal positions on abortion because it can be considered a reductio of their defence of abortion. Here I defend the liberal stance on abortion by arguing that the argument for the moral permissibility of killing newborns on ground of the social, psychological and economic burden on the parents recently put forward by Giubilini and Minerva is not valid; this is because they fail to show that newborns cannot be harmed and because there are morally relevant differences between fetuses and newborns...
May 2013: Journal of Medical Ethics
https://www.readbyqxmd.com/read/12917902/excitatory-amino-acid-antagonists-for-acute-stroke
#3
REVIEW
K W Muir, K R Lees
BACKGROUND: Focal cerebral ischaemia causes release of excitatory amino acid (EAA) neurotransmitters, principally glutamate, with resultant over-stimulation of EAA receptors and downstream pathways. Excess glutamate release is a pivotal event in the evolution of irreversible ischaemic damage in animal models of ischaemia, and drugs that modulate glutamate action either by inhibiting its release, or blocking post-synaptic receptors, are potent neuroprotective agents. Many clinical trials with EAA modulating drugs have been conducted, none individually demonstrating efficacy...
2003: Cochrane Database of Systematic Reviews
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