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negative pressure pulmonary edema

Deise M Pacheco, Viviane D Silveira, Alex Thomaz, Ramiro B Nunes, Viviane R Elsner, Pedro Dal Lago
OBJECTIVE: To analyze respiratory mechanics and hemodynamic alterations in an experimental model of chronic heart failure (CHF) following myocardial infarction. METHOD: Twenty-seven male adult Wistar rats were randomized to CHF group (n=12) or Sham group (n=15). Ten weeks after coronary ligation or sham surgery, the animals were anesthetized and submitted to respiratory mechanics and hemodynamic measurements. Pulmonary edema as well as cardiac remodeling were measured...
July 2016: Brazilian Journal of Physical Therapy
Priyanka Pandey, Zahara Ali, Ghulam Mohammad, M A Qadar Pasha
Biomarkers are essential to unravel the locked pathophysiology of any disease. This study investigated the role of biomarkers and their interactions with each other and with the clinical parameters to study the physiology of high-altitude pulmonary edema (HAPE) in HAPE-patients (HAPE-p) against adapted highlanders (HLs) and healthy sojourners, HAPE-controls (HAPE-c). For this, seven circulatory biomarkers, namely, epinephrine, norepinephrine, tyrosine hydroxylase, transforming growth factor beta 1, tumor necrosis factor alpha (TNFα), platelet-derived growth factor beta beta, and C-reactive protein (CRP), were measured in blood plasma of the three study groups...
2016: Therapeutics and Clinical Risk Management
Alfred A Bove
Although the lungs are a critical component of exercise performance, their response to exercise and other environmental stresses is often overlooked when evaluating pulmonary performance during high workloads. Exercise can produce capillary leakage, particularly when left atrial pressure increases related to left ventricular (LV) systolic or diastolic failure. Diastolic LV dysfunction that results in elevated left atrial pressure during exercise is particularly likely to result in pulmonary edema and capillary hemorrhage...
April 2016: Methodist DeBakey Cardiovascular Journal
Lisa Baumann, Susanne Haen, Christoph Berg, Ferruh Artunc, Reimer Riessen, Werner Spengler, Falko Fend, Michael Haap
HISTORY AND ADMISSION FINDINGS: We report on a 48-year-old man presenting with progressive hepatopathy and encephalopathy for two weeks based on a chronic hepatitis C. He takes ledipasvir and sofosbuvir (Harvoni) and ribavirin for almost 24 weeks. After admission to hospital his state deteriorated rapidly. He is directly transferred to the medical intensive care unit, where he died on day 3. INVESTIGATIONS: During the physical examination, a pronounced jaundice and significant peripheral edema were found...
July 2016: Deutsche Medizinische Wochenschrift
Jared W Meeker, Amy L Jaeger, William P Tillis
Reexpansion pulmonary edema (RPE) is a rare complication that can occur after rapid reinflation of the lung following thoracentesis of a pleural effusion or chest tube drainage of pneumothorax. The severity in clinical presentation can be widely varied from radiographic changes only to rapidly progressive respiratory failure requiring mechanical ventilation. The quick nature of onset and potential for serious decline in a previously stable patient makes it important to prepare, recognize, diagnose, and appropriately manage patients who develop RPE...
2016: Journal of Community Hospital Internal Medicine Perspectives
Sahar M Siddik-Sayyid, Waseem AlFahel, Mohamad F El-Khatib
Negative pressure pulmonary edema is a potentially life-threatening condition that may occur when a large negative intrathoracic pressure is generated against a 'physically' obstructed upper airway during emergence from anesthesia. We report a 35 year old male patient who is morbidly obese and undergoing laparoscopic gastric bypass who developed negative pressure pulmonary edema without any evidence of a 'physical' upper airway obstruction. In our patient, the negative pressure pulmonary edema occurred after complete reversal of neuromuscular blockade and during manual positive pressure ventilation with the endotracheal tube still in place and in the presence of an oral airway...
February 2016: Middle East Journal of Anesthesiology
Zahara Ali, Mohammad Waseem, Rahul Kumar, Priyanka Pandey, Ghulam Mohammad, M A Qadar Pasha
CONTEXT: Few potential candidate genes coding for type I and II receptors of transforming growth factor beta signaling pathway and the serotonin transporter have been associated with pulmonary hypertension (PH). The latter being a phenotype for high altitude pulmonary edema (HAPE), these genes are hypothesized to be crucial markers to investigate under the hypobaric hypoxic environment of high altitude. AIMS: We hence aimed to investigate bone-morphogenetic protein-2 (BMP2), bone morphogenetic protein receptor type-II (BMPR-2), activin receptor-like kinase-1 (ALK-1), serotonin transporter (5-HTT) and serotonin (5-HT) for their contribution, individually/epistatically, to clinical endpoints by altering downstream signaling molecules...
August 22, 2016: Gene
Eric P Cantey, James M Walter, Thomas Corbridge, Jeffrey H Barsuk
PURPOSE OF REVIEW: Although thoracentesis is generally considered safe, procedural complications are associated with increased morbidity, mortality, and healthcare costs. In this article, we review the risk factors and prevention of the most common complications of thoracentesis including pneumothorax, bleeding (chest wall hematoma and hemothorax), and re-expansion pulmonary edema. RECENT FINDINGS: Recent data support the importance of operator expertise and the use of ultrasound in reducing the risk of iatrogenic pneumothorax...
July 2016: Current Opinion in Pulmonary Medicine
Deise M Pacheco, Viviane D Silveira, Alex Thomaz, Ramiro B Nunes, Viviane R Elsner, Pedro Dal Lago
Objective To analyze respiratory mechanics and hemodynamic alterations in an experimental model of chronic heart failure (CHF) following myocardial infarction. Method Twenty-seven male adult Wistar rats were randomized to CHF group (n=12) or Sham group (n=15). Ten weeks after coronary ligation or sham surgery, the animals were anesthetized and submitted to respiratory mechanics and hemodynamic measurements. Pulmonary edema as well as cardiac remodeling were measured. Results The CHF rats showed pulmonary edema 26% higher than the Sham group...
April 8, 2016: Brazilian Journal of Physical Therapy
Mallar Bhattacharya, Richard H Kallet, Lorraine B Ware, Michael A Matthay
Negative-pressure pulmonary edema (NPPE) or postobstructive pulmonary edema is a well-described cause of acute respiratory failure that occurs after intense inspiratory effort against an obstructed airway, usually from upper airway infection, tumor, or laryngospasm. Patients with NPPE generate very negative airway pressures, which augment transvascular fluid filtration and precipitate interstitial and alveolar edema. Pulmonary edema fluid collected from most patients with NPPE has a low protein concentration, suggesting hydrostatic forces as the primary mechanism for the pathogenesis of NPPE...
October 2016: Chest
Ramesh Venkatesh, Preety Gautam, Parul Dutta, Preeti Bala
BACKGROUND: Negative pressure pulmonary edema is a potentially life-threatening complication after general anesthesia in young healthy individuals that results from upper airway obstruction followed by strong inspiratory effort. It is a known complication after nasal or upper airway surgery. Occurrence of such a life-threatening complication in an eye care setting where advanced intensive care is usually lacking is rare. CASE PRESENTATION: A 15-year-old Asian boy presented to our hospital with a penetrating eye injury caused by a pellet...
2016: Journal of Medical Case Reports
Yazeed Toukan, Michal Gur, Lea Bentur
A 12-year-old boy developed severe acute respiratory distress during a school break requiring resuscitative measures. The episode started shortly after a short choking episode with a cookie. History, physical examination, laboratory results, chest X-ray, and clinical course supported the diagnosis of negative pressure pulmonary edema (NPPE). NPPE occurring outside a hospital setting, especially following a short episode of choking on a cookie, is rarely reported in children. Understanding the pathophysiological mechanisms contributing to pulmonary edema can help in distinguishing NPPE from other causes of fulminant respiratory distress, and especially from other causes of noncardiogenic pulmonary edema...
July 2016: Pediatric Pulmonology
Eunkyung Choi, Junggu Yi, Younghoon Jeon
Postoperative negative pressure pulmonary edema (NPPE) is a rare, but well-known life-threatening complication of acute upper airway obstruction (UAO) which develops after general anesthesia. The pronounced inspiratory efforts following UAO lead to excessive negative inspiratory pressure, which may cause acute pulmonary edema. Early recognition and prompt treatment of NPPE is necessary to prevent patient morbidity and mortality. In addition, the physician should carefully manage the patient who has risk factors of UAO to prevent this situation...
2015: International Medical Case Reports Journal
Lais Bastos Pessanha, Adriana Maria Fonseca de Melo, Flavia Silva Braga, Gabriel Antonio de Oliveira, Livia Guidoni de Assis Barbosa, Antonio Roberto Carrareto
No abstract text is available yet for this article.
May 2015: Radiologia Brasileira
Min Zhou, Ji Dai, Min Du, Wei Wang, Changxing Guo, Yi Wang, Rui Tang, Fengling Xu, Zhuqing Rao, Gengyun Sun
BACKGROUND: The role of dobutamine in the relief of pulmonary edema during septic shock-induced acute respiratory distress syndrome (ARDS) remains undetermined, due to a lack of controllable and quantitative clinical studies. Our objective was to assess the potential effects of dobutamine on extravascular lung water index (ELWI) in septic shock-induced ARDS, reflecting its importance in pulmonary edema. At the same time, ventilator function and perfusion parameters were evaluated. METHODS: We designed a prospective, non-randomized, non-blinded, controlled study to compare the differences in PiCCO parameters after 6 h of constant dobutamine infusion (15 μg/kg/min), in the baseline parameters in 26 septic shock-related ARDS patients with cardiac index ≥ 2...
August 2016: Artificial Cells, Nanomedicine, and Biotechnology
Mengchan Ou, Zhen Luo, Juan Liu, Yaoxin Yang, Yu Li
We describe a case of negative pressure pulmonary edema that occurred in the post-anesthesia care unit in a patient who had undergone transsphenoidal resection of a pituitary adenoma. Negative pressure pulmonary edema is an uncommon complication of general anesthesia. In this case, the patient's nasal cavity had been filled with surgical packs and she had not become accustomed to breathing through her mouth, in addition to her large tongue and small oropharyngeal cavity, residual effect of anesthetic may resulting in tongue falling which caused airway obstruction...
2015: International Journal of Clinical and Experimental Medicine
Ahmed M Maged, Noura ElNassery, Mona Fouad, Aly Abdelhafiz, Walaa Al Mostafa
OBJECTIVE: To evaluate the association between uterine artery Doppler measurements and maternal complications among women with severe pre-eclampsia. METHODS: As part of a cross-sectional study, women with a single intrauterine pregnancy of more than 28 weeks and a diagnosis of severe pre-eclampsia were enrolled at a unit in Cairo, Egypt, between December 2012 and September 2014. Uterine artery Doppler was evaluated and maternal complications were recorded. RESULTS: Among the 100 participants, 76 (76%) experienced maternal complications...
October 2015: International Journal of Gynaecology and Obstetrics
Jae Jun Kim, Yong Hwan Kim, Si Young Choi, Seong Cheol Jeong, Seok Whan Moon
Reexpansion pulmonary edema is a rare but potentially life-threatening condition that occurs when a collapsed lung reexpands, usually in the same side of collapsed lung. We present a rare case in which a 57-year-old Korean man had a large amount of malignant pleural effusion. After undergoing tube thoracostomy drainage for the pleural effusion, a contralateral reexpansion pulmonary edema developed while the ipsilateral lung was half collapsed. The patient was dyspneic with an oxygen saturation that dropped to 66 %...
2015: Journal of Cardiothoracic Surgery
Yasuko Ikeda-Miyagawa, Takako Kihara, Rumiko Matsuda
We experienced a case of negative pressure pulmonary edema (NPPE) after administration of sugammadex under general anesthesia with LMA Supreme. Sugammadex was administered to a 76-year-old female patient following spontaneous ventilation. Immediately after the administration of sugammadex, laryngospasm occurred, and the patient's oxygen saturation dropped to 70%. About five minutes after the appearance of laryngospasm, she started to breathe spontaneously. However, a chest x-ray at this time showed pulmonary edema We suspected that sugammadex administration had triggered the laryngospasm, causing postoperative NPPE...
December 2014: Masui. the Japanese Journal of Anesthesiology
William F Weitzel, James Hamilton, Xianglong Wang, Joseph L Bull, Alan Vollmer, April Bowman, Jonathan Rubin, Grant H Kruger, Jing Gao, Michael Heung, Panduranga Rao
BACKGROUND/AIMS: Recently, ultrasound signals termed 'lung water comets' associated with pulmonary edema have been correlated with adverse clinical events in dialysis patients. These comets fluctuate substantially during the ultrasound exam highlighting the need for objective quantitative measurement methods. METHODS: We developed an image-processing algorithm for the detection and quantification of lung comets. Quantification measures included comet number (comet count) and the fraction of the ultrasound beams with comet findings (comet fraction)...
2015: Blood Purification
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