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Derm infectious

Katie Sell, Brian Hainline, Michael Yorio, Mark Kovacs
OBJECTIVE: To examine the incidence of illness and highlight gender differences in tennis players competing in a major professional tennis tournament over a 16-year period between 1994 and 2009. DESIGN: Descriptive epidemiology study of illness trends in professional tennis players. SETTING: Archival data from the US Open Tennis Championships. PARTICIPANTS: Participants in the US Open Tennis Championships main draw from 1994 to 2009...
January 2013: Clinical Journal of Sport Medicine: Official Journal of the Canadian Academy of Sport Medicine
Rie Hasebe, Michihito Sasaki, Hirofumi Sawa, Ryuichi Wada, Takashi Umemura, Takashi Kimura
We investigated the mechanism by which equine herpesvirus-1 (EHV-1) enters primary cultured equine brain microvascular endothelial cells (EBMECs) and equine dermis (E. Derm) cells. EHV-1 colocalized with caveolin in EBMECs and the infection was greatly reduced by the expression of a dominant negative form of equine caveolin-1 (ecavY14F), suggesting that EHV-1 enters EBMECs via caveolar endocytosis. EHV-1 entry into E. Derm cells was significantly reduced by ATP depletion and treatments with lysosomotropic agents...
October 25, 2009: Virology
B C H Ang, M I C Chen, T L H Goh, Y Y Ng, S W Fan
INTRODUCTION: A common approach to the surveillance of emerging infectious diseases and agents of bioterrorism is to analyse electronically captured data for disease syndromes. The Patient Care Enhancement System (PACES) is a form of electronic medical records presently in service in the Singapore Armed Forces (SAF). We assess the feasibility of PACES data for surveillance, describe time-trends, and identify methods of sub-analysis which could improve performance. MATERIALS AND METHODS: Medical consults from July 2000 to June 2003 were extracted...
October 2005: Annals of the Academy of Medicine, Singapore
J S Rhim, H S Ro, E B Kim, R V Gilden, R J Huebner
A horse skin cell line (E. Derm, NBL-6, CCL-57) was susceptible to focus formation by the Kirsten mouse sarcoma virus, feline sarcoma virus (ST stain) and the MSV pseudotypes with woolly monkey, gibbon monkey, RD-114, AT-124, baboon placenta and murine xenotropic (BALB/c 3T3 and C57L/JD) type-C viruses. Foci were detected within 5 days after infection and the transformed cells continued to produce infectious virus and group-specific antigen of their respective type-C leukemia viruses. The transformation efficiency of various type-C sarcoma viruses in horse cells was also very high...
February 15, 1975: International Journal of Cancer. Journal International du Cancer
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