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intestine mucus dopamine

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https://www.readbyqxmd.com/read/20821402/human-laryngeal-ganglia-contain-both-sympathetic-and-parasympathetic-cell-types
#1
Marta Ibanez, Francisco J Valderrama-Canales, Eva Maranillo, Teresa Vazquez, Arán Pascual-Font, Stephen McHanwell, Jose Sanudo
The presence of ganglia associated with the laryngeal nerves is well documented. In man, these ganglia have been less well studied than in other species and, in particular, the cell types within these ganglia are less well characterized. Using a panel of antibodies to a variety of markers found in the paraganglion cells of other species, we were able to show the existence of at least two populations of cells within human laryngeal paraganglia. One population contained chromogranin and tyrosine hydroxylase representing a neurosecretory population possibly secreting dopamine...
September 2010: Clinical Anatomy
https://www.readbyqxmd.com/read/12601282/dopamine-induced-protection-against-indomethacin-evoked-intestinal-lesions-in-rats-role-of-anti-intestinal-motility-mediated-by-d2-receptors
#2
Tohru Miyazawa, Masao Matsumoto, Shinichi Kato, Koji Takeuchi
BACKGROUND: Although it is known that dopamine prevents various gastrointestinal lesions, the underlying mechanism remains unclear. In the present study, we examined the protective effect of dopamine on indomethacin-induced small intestinal lesions, in relation to intestinal hypermotility. MATERIAL/METHODS: Male SD rats received indomethacin (10 mg/kg) subcutaneously (s.c.), and the small intestine was examined for lesions 24 hr later. Dopamine (1-10 mg/kg) or atropine (3 mg/kg) was administered s...
February 2003: Medical Science Monitor: International Medical Journal of Experimental and Clinical Research
https://www.readbyqxmd.com/read/9558036/gastroduodenal-mucosal-protection-induced-by-dehydroleucodine-mucus-secretion-and-role-of-monoamines
#3
COMPARATIVE STUDY
A B Penissi, T H Fogal, J A Guzmán, R S Piezzi
In previous work we have demonstrated that dehydroleucodine (DhL) prevents gastric damage induced by necrosis-inducing agents such as absolute ethanol (EtOH). In this study we examine the effects of DhL on gastroduodenal morphology and monoamine levels by histological and biochemical methods, respectively, as an approach to elucidating the cytoprotective mechanism of the drug. Histological evidence shows that DhL prevents formation of gastroduodenal mucosal lesions induced by EtOH and that this protective effect is related to the ability of the drug to stimulate mucus production...
April 1998: Digestive Diseases and Sciences
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