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Excessive sweating high cortisol

Katharina M Gross, Andrea B Schote, Katja Kerstin Schneider, André Schulz, Jobst Meyer
Primary hyperhidrosis is defined as excessive sweating of certain body areas without physiological reasons. Hyperhidrotic individuals report a high psychological strain and an impairment of their quality of life. Thus, the aim of the study is to investigate the relation between hyperhidrosis and different psychological as well as physiological aspects of chronic stress as a co-factor for the etiology of depression. In this study, forty hyperhidrotic subjects were compared to forty age- and sex-matched healthy control subjects...
2014: PloS One
F Bayraktar, L Kebapcilar, M A Kocdor, S L Asa, S Yesil, S Canda, T Demir, A Saklamaz, M Seçil, B Akinci, S Yener, A Comlekci
Ectopic production of corticotropin-releasing hormone (CRH) by a pheochromocytoma is an infrequent cause of Cushing's syndrome. We report the case of a 43-year-old man with Cushing's syndrome due to a CRH-producing adrenal pheochromocytoma. The patient had clinical and biochemical evidence of hypercortisolism in conjunction with high ACTH levels and non-suppressible serum cortisol levels on low-dose and high-dose dexamethasone suppression testing. In addition to these clinical features of one month's duration, the patient developed symptoms of pheochromocytoma including headache, hypertension that was resistant to conventional therapy and excessive sweating...
September 2006: Experimental and Clinical Endocrinology & Diabetes
Brigitte Bocchi, Sabine Kenouch, Maxime Lamarre-Cliche, Martine Muffat-Joly, Michel Hubert Capron, Jean Fiet, Gilles Morineau, Michel Azizi, Jean Pierre Bonvalet, Nicolette Farman
The enzyme 11-beta hydroxysteroid dehydrogenase type 2 plays a major role in blood pressure regulation. It metabolizes glucocorticoid hormones into derivatives with low affinity for the mineralocorticoid receptor, preventing its permanent occupancy by circulating cortisol, which is 100- to 1000-fold more abundant than aldosterone in the plasma. Inactivating mutations of the enzyme result in severe hypertension, as seen in children with apparent mineralocorticoid excess syndrome. In patients with essential hypertension, however, attempts to evidence enzyme deficiency have been inconclusive...
April 2004: Hypertension
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