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vestibular paroxysmia

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https://www.readbyqxmd.com/read/29928252/susceptibility-to-fear-of-heights-in-bilateral-vestibulopathy-and-other-disorders-of-vertigo-and-balance
#1
Thomas Brandt, Eva Grill, Michael Strupp, Doreen Huppert
Aims: To determine the susceptibility to visual height intolerance (vHI) in patients with acquired bilateral vestibulopathy (BVP). The question was whether postural instability in BVP, which is partially compensated for by visual substitution of the impaired vestibular control of balance, leads to an increased susceptibility. This is of particular importance since fear of heights is dependent on body posture, and visual control of balance at heights can no longer substitute vestibular input. For comparison susceptibility to vHI was determined in patients with other vestibular or functional disorders...
2018: Frontiers in Neurology
https://www.readbyqxmd.com/read/29924039/successive-occurrence-of-vertebrobasilar-dolichectasia-induced-trigeminal-neuralgia-vestibular-paroxysmia-and-hemifacial-spasm-a-case-report
#2
Jingzhe Han, Tingting Wang, Yanan Xie, Duanhua Cao, Zhilei Kang, Xueqin Song
RATIONALE: Study reported an extremely rare case of trigeminal neuralgia, vestibular paroxysmia, and hemifacial spasm successively occurring in a patient with vertebrobasilar dolichoectasia (VBD). PATIENT CONCERNS: A 66-year-old female patient presented with episodic vertigo for 20 days before she was admitted to our hospital. Vertigo suddenly occurred when the patient rotated her head 20 days ago, the symptoms of vertigo were improved after continuous 1 to 3 seconds, and similar symptoms were repeated in sleep and rest, with a frequency of 30 to 40 times per day...
June 2018: Medicine (Baltimore)
https://www.readbyqxmd.com/read/29910862/vestibular-paroxysmia-in-vestibular-neuritis-a-case-report
#3
Michele Ori, Valeria Gambacorta, Giampietro Ricci, Mario Faralli
The term vestibular paroxysmia (VP) was introduced for the first time by Brandt and Dieterich in 1994. In 2016, the Barany Society formulated the International Classification of VP, focusing in particular on the number and duration of attacks, on the differential diagnosis and on the therapy. Ephaptic discharges in the proximal part of the eighth cranial nerve, which is covered by oligodendrocytes, are assumed to be the neural basis of VP. We report the first case in literature of an onset of symptoms and signs typical of VP in a young man following acute unilateral vestibular loss not combined with auditory symptoms...
March 6, 2018: Audiology Research
https://www.readbyqxmd.com/read/29747309/-etiological-analysis-on-patients-with-vertigo-or-dizziness
#4
H Xue, Y Chong, Z D Jiang, Z L Liu, L Ding, S L Yang, L Wang, W P Xiang
Objective: To explore the spectrum of causes for patients with vertigo or dizziness in the Department of Neurology, and provide a reference for diagnosis and treatment of patients with vertigo or dizziness. Methods: Clinical data of patients in the Department of Neurology, Baotou Central Hospital between January 2016 and September 2017 was retrospectively analyzed. The target group under study was diagnosed based on the uniform diagnostic criteria. Results: A total of 9 200 patients with a chief complaint of vertigo or dizziness were included...
April 24, 2018: Zhonghua Yi Xue za Zhi [Chinese medical journal]
https://www.readbyqxmd.com/read/29695895/randomized-trial-of-betahistine-mesilate-tablets-as-augmentation-for-oxcarbazepine-and-carbamazepine-in-treating-vestibular-paroxysmia
#5
RANDOMIZED CONTROLLED TRIAL
Hui Xue, Wenping Xiang, Yichuan Yu, Guorong Liu, Yi Chong, Jiying Zhou
Background: Vestibular paroxysmia (VP) is a rare episodic peripheral vestibular disorder. This study was conducted to compare the efficacy and acceptability of carbamazepine (CBZ) plus betahistine mesilate tablets (BMT) (CBZ+BMT) and oxcarbazepine (OXC) plus BMT (OXC+BMT) in treating VP, and investigated whether the synergistic effect could be increased along with the increased dose of BMT. Methods: VP patients were recruited and randomly assigned to receive CBZ+BMT or OXC+BMT...
2018: Drug Design, Development and Therapy
https://www.readbyqxmd.com/read/29301160/-pharmacotherapy-of-vestibular-disorders-nystagmus-and-cerebellar-disorders
#6
K Feil, N Böttcher, O Kremmyda, C Muth, J Teufel, A Zwergal, T Brandt, M Strupp
There are currently different groups of drugs for the pharmacotherapy of vertigo, nystagmus and cerebellar disorders: antiemetics; anti-inflammatories, antimenieres, and antimigraineous medications and antidepressants, anticonvulsants, aminopyridines as well as acetyl-DL-leucine. In acute unilateral vestibulopathy, corticosteroids improve the recovery of peripheral vestibular function, but currently there is not sufficient evidence for a general recommendation. There is insufficient evidence to support the view that 16 mg t...
January 2018: Laryngo- Rhino- Otologie
https://www.readbyqxmd.com/read/29282702/treatment-of-dizziness-an-interdisciplinary-update
#7
REVIEW
Rainer Spiegel, Heiko Rust, Thomas Baumann, Hergen Friedrich, Raoul Sutter, Martina Göldlin, Christiane Rosin, René Müri, Georgios Mantokoudis, Roland Bingisser, Michael Strupp, Roger Kalla
This review provides an update on interdisciplinary treatment for dizziness. Dizziness can have various causes and the treatment offered should depend on the cause. After reading this article, the clinician will have an overview of current treatment recommendations. Recommendations are made for the most prevalent causes of dizziness including acute and chronic vestibular syndromes, vestibular neuritis, benign paroxysmal positional vertigo, endolymphatic hydrops and Menière’s disease, vestibular paroxysmia and vestibular migraine, cardiac causes, transient ischaemic attacks and strokes, episodic ataxia type 2, persistent postural-perceptual dizziness, bilateral vestibulopathy, degenerative, autoimmune and neoplastic diseases, upbeat- and downbeat nystagmus...
2017: Swiss Medical Weekly
https://www.readbyqxmd.com/read/29204964/a-randomized-double-blind-placebo-controlled-cross-over-trial-vestparoxy-of-the-treatment-of-vestibular-paroxysmia-with-oxcarbazepine
#8
RANDOMIZED CONTROLLED TRIAL
Otmar Bayer, Tatiana Brémová, Michael Strupp, Katharina Hüfner
OBJECTIVE: Vestibular paroxysmia (VP) is characterized by short, often oligosymptomatic attacks of vertigo which occur spontaneously or are sometimes provoked by turning the head. Despite the description of the disease almost 40 years ago (first termed "disabling positional vertigo"), no controlled treatment trial has been published to date. The Vestparoxy trial was designed as a randomized, placebo-controlled, double-blind cross-over trial to examine the therapeutic effect of oxcarbazepine (OXA) in patients with definite or probable VP...
February 2018: Journal of Neurology
https://www.readbyqxmd.com/read/28395427/-etiological-analysis-on-patients-in-department-of-vertigo-and-dizziness-oriented-outpatient
#9
F Li, X G Wang, J H Zhuang, Y Chen, X W Zhou, B Gao, H H Gu
Objective: We aimed to explore the spectrum of causes for patients in department of vertigo and dizziness oriented outpatient, in order to provide a reference for diagnosis and treatment of patients with vertigo or dizziness. Methods: Retrospective analysis were carried out with clinical data of patients in our department of vertigo and dizziness oriented outpatient. The target group under study was diagnosed based on the uniform diagnostic criteria, and re-visiting patients were excluded. Results: This clinical study was conducted on 5 348 cases, who visited our vertigo and dizziness oriented outpatient from December 2012 to July 2015...
April 11, 2017: Zhonghua Yi Xue za Zhi [Chinese medical journal]
https://www.readbyqxmd.com/read/28262641/vestibular-paroxysmia-diagnostic-criteria
#10
Michael Strupp, Jose A Lopez-Escamez, Ji-Soo Kim, Dominik Straumann, Joanna C Jen, John Carey, Alexandre Bisdorff, Thomas Brandt
This paper describes the diagnostic criteria for vestibular paroxysmia (VP) as defined by the Classification Committee of the Bárány Society. The diagnosis of VP is mainly based on the patient history and requires: A) at least ten attacks of spontaneous spinning or non-spinning vertigo; B) duration less than 1 minute; C) stereotyped phenomenology in a particular patient; D) response to a treatment with carbamazepine/oxcarbazepine; and F) not better accounted for by another diagnosis. Probable VP is defined as follows: A) at least five attacks of spinning or non-spinning vertigo; B) duration less than 5 minutes; C) spontaneous occurrence or provoked by certain head-movements; D) stereotyped phenomenology in a particular patient; E) not better accounted for by another diagnosis...
2016: Journal of Vestibular Research: Equilibrium & Orientation
https://www.readbyqxmd.com/read/28197658/-vestibular-paroxysmia-and-paroxysmal-tinnitus
#11
P P Urban, R Brüning
No abstract text is available yet for this article.
February 2018: Der Nervenarzt
https://www.readbyqxmd.com/read/28002135/functional-dizziness-from-phobic-postural-vertigo-and-chronic-subjective-dizziness-to-persistent-postural-perceptual-dizziness
#12
Marianne Dieterich, Jeffrey P Staab
PURPOSE OF REVIEW: Functional dizziness is the new term for somatoform or psychogenic dizziness. The aim of this study is to review arguments for the new nomenclature, clinical features, possible pathomechanisms, and comorbidities of functional dizziness. RECENT FINDINGS: The prevalence of functional dizziness as a primary cause of vestibular symptoms amounts to 10% in neuro-otology centers. Rates of psychiatric comorbidity in patients with structural vestibular syndromes are much higher with nearly 50% and with highest rates in patients with vestibular migraine, vestibular paroxysmia, and Ménière's disease...
December 20, 2016: Current Opinion in Neurology
https://www.readbyqxmd.com/read/28002123/functional-dizziness-from-phobic-postural-vertigo-and-chronic-subjective-dizziness-to-persistent-postural-perceptual-dizziness
#13
REVIEW
Marianne Dieterich, Jeffrey P Staab
PURPOSE OF REVIEW: Functional dizziness is the new term for somatoform or psychogenic dizziness. The aim of this study is to review arguments for the new nomenclature, clinical features, possible pathomechanisms, and comorbidities of functional dizziness. RECENT FINDINGS: The prevalence of functional dizziness as a primary cause of vestibular symptoms amounts to 10% in neuro-otology centers. Rates of psychiatric comorbidity in patients with structural vestibular syndromes are much higher with nearly 50% and with highest rates in patients with vestibular migraine, vestibular paroxysmia, and Ménière's disease...
February 2017: Current Opinion in Neurology
https://www.readbyqxmd.com/read/27306762/therapy-of-vestibular-paroxysmia-superior-oblique-myokymia-and-ocular-neuromyotonia
#14
REVIEW
Michael Strupp, Marianne Dieterich, Thomas Brandt, Katharina Feil
Neurovascular compression syndromes are characterized by recurrent attacks of neurological symptoms and clinical signs depending on the cranial nerve affected. It is assumed that pulsatile compression of the nerve is caused mainly by an artery. The result is segmental demyelination of the transition zone or the central part of the cranial nerve, which is covered by oligodendrocytes, and subsequent ephaptic axonal transmission. Compression of the vestibular nerve can cause attacks of spinning or non-spinning vertigo: vestibular paroxysmia...
July 2016: Current Treatment Options in Neurology
https://www.readbyqxmd.com/read/27158666/tinnitus-oscillopsia-and-hyperventilation-induced-nystagmus-vestibular-paroxysmia
#15
Bryan K Ward, Daniel R Gold
Vestibular paroxysmia is the name given to vascular compression of the vestibulocochlear nerve. Substantial evidence has been discovered in support of vascular compression of the trigeminal nerve as the etiology for trigeminal neuralgia, and effective therapies have been targeted to address this pathophysiology. Perhaps due to the common and often vaguely-described symptoms of dizziness and tinnitus, vascular compression of the vestibulocochlear nerve as a cause of symptoms has remained controversial. Recent clinical studies, however, have better defined diagnostic criteria for vestibular paroxysmia...
2016: Open Journal of Clinical & Medical Case Reports
https://www.readbyqxmd.com/read/27083889/vestibular-paroxysmia-a-treatable-neurovascular-cross-compression-syndrome
#16
REVIEW
Thomas Brandt, Michael Strupp, Marianne Dieterich
The leading symptoms of vestibular paroxysmia (VP) are recurrent, spontaneous, short attacks of spinning or non-spinning vertigo that generally last less than one minute and occur in a series of up to 30 or more per day. VP may manifest when arteries in the cerebellar pontine angle cause a segmental, pressure-induced dysfunction of the eighth nerve. The symptoms are usually triggered by direct pulsatile compression with ephaptic discharges, less often by conduction blocks. MR imaging reveals the neurovascular compression of the eighth nerve (3D constructive interference in steady state and 3D time-of-flight sequences) in more than 95% of cases...
April 2016: Journal of Neurology
https://www.readbyqxmd.com/read/27056408/efficacy-and-acceptability-of-oxcarbazepine-vs-carbamazepine-with-betahistine-mesilate-tablets-in-treating-vestibular-paroxysmia-a-retrospective-review
#17
COMPARATIVE STUDY
Chong Yi, Xiang Wenping, Xue Hui, He Xin, Li Xiue, Zhang Jun, Geng Shangyong
OBJECTIVES: Vestibular paroxysmia (VP) is a rare episodic peripheral vestibular disorder that can cause acute short attacks of vertigo. This study aimed to compare the efficacy and acceptability of carbamazepine (CBZ), CBZ plus betahistine mesilate tablets (BMT) and oxcarbazepine (OXC) plus BMT in treating VP within 12 weeks. METHODS: A retrospective analysis of data from 196 VP patients treated in our hospital was conducted. There were 73 patients receiving CBZ, 65 patients receiving CBZ+BMT and 58 patients receiving OXC+BMT...
June 2016: Postgraduate Medicine
https://www.readbyqxmd.com/read/26892985/imaging-of-neurovascular-compression-syndromes-trigeminal-neuralgia-hemifacial-spasm-vestibular-paroxysmia-and-glossopharyngeal-neuralgia
#18
REVIEW
S Haller, L Etienne, E Kövari, A D Varoquaux, H Urbach, M Becker
Neurovascular compression syndromes are usually caused by arteries that directly contact the cisternal portion of a cranial nerve. Not all cases of neurovascular contact are clinically symptomatic. The transition zone between the central and peripheral myelin is the most vulnerable region for symptomatic neurovascular compression syndromes. Trigeminal neuralgia (cranial nerve V) has an incidence of 4-20/100,000, a transition zone of 4 mm, with symptomatic neurovascular compression typically proximal. Hemifacial spasm (cranial nerve VII) has an incidence of 1/100,000, a transition zone of 2...
August 2016: AJNR. American Journal of Neuroradiology
https://www.readbyqxmd.com/read/26643594/-peripheral-central-and-functional-vertigo-syndromes
#19
REVIEW
M Strupp, M Dieterich, A Zwergal, T Brandt
Depending on the temporal course, three forms of vertigo syndrome can be differentiated: 1) vertigo attacks, e.g. benign paroxysmal positional vertigo (BPPV), Menière's disease and vestibular migraine, 2) acute spontaneous vertigo lasting for days, e.g. acute unilateral vestibulopathy, brainstem or cerebellar infarction and 3) symptoms lasting for months or years, e.g. bilateral vestibulopathy and functional vertigo. The specific therapy of the various syndromes is based on three principles: 1) physical treatment with liberatory maneuvers for BPPV and balance training for vestibular deficits, 2) pharmacotherapy, e...
December 2015: Der Nervenarzt
https://www.readbyqxmd.com/read/26421856/-pharmacotherapy-of-vestibular-disorders-nystagmus-and-cerebellar-disorders
#20
REVIEW
K Feil, N Böttcher, O Kremmyda, C Muth, J Teufel, A Zwergal, T Brandt, M Strupp
There are currently different groups of drugs for the pharmacotherapy of vertigo, nystagmus and cerebellar disorders: antiemetics; anti-inflammatories, antimenieres, and antimigraineous medications and antidepressants, anticonvulsants, aminopyridines as well as acetyl-DL-leucine. In acute unilateral vestibulopathy, corticosteroids improve the recovery of peripheral vestibular function, but currently there is not sufficient evidence for a general recommendation. There is insufficient evidence to support the view that 16 mg t...
September 2015: Fortschritte der Neurologie-Psychiatrie
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