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Turner syndrome electrolyte imbalance

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https://www.readbyqxmd.com/read/23720520/pathophysiology-of-white-nose-syndrome-in-bats-a-mechanistic-model-linking-wing-damage-to-mortality
#1
Lisa Warnecke, James M Turner, Trent K Bollinger, Vikram Misra, Paul M Cryan, David S Blehert, Gudrun Wibbelt, Craig K R Willis
White-nose syndrome is devastating North American bat populations but we lack basic information on disease mechanisms. Altered blood physiology owing to epidermal invasion by the fungal pathogen Geomyces destructans (Gd) has been hypothesized as a cause of disrupted torpor patterns of affected hibernating bats, leading to mortality. Here, we present data on blood electrolyte concentration, haematology and acid-base balance of hibernating little brown bats, Myotis lucifugus, following experimental inoculation with Gd...
August 23, 2013: Biology Letters
https://www.readbyqxmd.com/read/23568916/electrolyte-depletion-in-white-nose-syndrome-bats
#2
Paul M Cryan, Carol Uphoff Meteyer, David S Blehert, Jeffrey M Lorch, DeeAnn M Reeder, Gregory G Turner, Julie Webb, Melissa Behr, Michelle Verant, Robin E Russell, Kevin T Castle
The emerging wildlife disease white-nose syndrome is causing widespread mortality in hibernating North American bats. White-nose syndrome occurs when the fungus Geomyces destructans infects the living skin of bats during hibernation, but links between infection and mortality are underexplored. We analyzed blood from hibernating bats and compared blood electrolyte levels to wing damage caused by the fungus. Sodium and chloride tended to decrease as wing damage increased in severity. Depletion of these electrolytes suggests that infected bats may become hypotonically dehydrated during winter...
April 2013: Journal of Wildlife Diseases
https://www.readbyqxmd.com/read/4020468/fluid-balance-and-secretion-of-antidiuretic-hormone-following-transsphenoidal-pituitary-surgery-a-preliminary-series
#3
S J Whitaker, C I Meanock, G F Turner, P J Smythe, J D Pickard, A R Noble, V Walker
Hyponatremia developing some days after transsphenoidal pituitary adenectomy is a treacherous complication of uncertain cause. Of 19 patients monitored in a pilot study at the Wessex Neurological Centre, plasma sodium fell below 125 mmol/liter in three patients at times ranging from 6 to 9 days postoperatively. One patient had evidence of inappropriate secretion of arginine vasopressin (AVP), and the other two probably had steroid insufficiency despite apparently adequate steroid cover. In a more detailed study, the fluid and sodium balance of a further 16 patients was monitored for 7 to 11 days following transsphenoidal surgery together with plasma cortisol, renin, and AVP concentrations...
September 1985: Journal of Neurosurgery
https://www.readbyqxmd.com/read/1116910/clinical-study-of-intersex-states-in-nigerian-children
#4
F A Nwako, Mbuagbaw
Early diagnosis of intersex is essential to avoid indecision and to establish the correct sex of rearing before mistakes have been made. Diagnosis of adrenogenital syndrome is singularly important for delay in diagnosis may lead to stunted growth or death from electrolyte imbalance. All others except the hormonally induced types are sterile, the gonad can be ignored and the sex of rearing is determined by the external genital configuration which can be most easily created by surgery plus hormones. Patients except those with maternal virilization may also need castration and hormone therapy corresponding with the chosen sex of rearing...
February 1975: International Surgery
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